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Hepatitis C-related atherosclerosis and cardiovascular disease.
Although the link between hepatitis C virus (HCV) infection and cardiovascular diseases is well-established, the precise mechanisms by which HCV infection may accelerate atherogenesis are still not completely understood. A wide array of factors may contribute to the progression of the disease and risk of cardiovascular events, such as, the host and viral factors (genotype, viral load, genetic background), the HCV treatment (treatment-induced sustained virologic response, non-response to therapy, complications during and after treatment), the co-morbidities associated with HCV infection (metabolic syndrome, diabetes mellitus, and obesity), and the host's immune response to the HCV infection. The increasing prevalence of HCV-associated liver disease and the increasing numbers of individuals undergoing HCV therapy suggest that cardiovascular disease will be an increasing problem for these patients. Hence, an improved understanding of the mechanisms underlying the interaction of HCV infection with the cardiovascular system is needed in order to develop strategies to reduce the impact of this disease 0b46394aab